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Finding answers on the web can be challenging, often requiring multiple attempts to obtain relevant results. While AI tools like ChatGPT, Perplexity, Grok, Deepseek, and Google Gemini strive to provide helpful responses, there may be instances where they cannot provide specific answers to your questions. Please remember this site has a search function (upper right) so that you can search for the content you are looking for. One Day MD isn’t about us—it’s about you. Can you be your own health advocate? You know your body best. While your doctor isn’t always there, you have constant access to resources like the internet, Google, and ChatGPT. It’s time to take control of your health. Learning from online resources won’t make you an MD overnight, but it’s a powerful first step toward better health for you and those you care about. The internet now provides access to global information and connections, empowering you to build a community or join a movement with ease. That said, you must als...
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Repositioning Anti-Parasitic Drugs for Cancer Treatment: Analyzing drugs like ivermectin, fenbendazole and mebendazole for their potential to disrupt cancer cell or enhance chemotherapy

By Editorial Team
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Antiparasite drugs such as ivermectin, fenbendazole, and mebendazole have shown promising potential in cancer therapy by disrupting cancer cell signaling and enhancing chemotherapy efficacy through multiple mechanisms. Fenbendazole Fenbendazole, a benzimidazole anthelmintic, exhibits several anti-cancer effects: Microtubule disruption:  Fenbendazole acts as a moderate microtubule destabilizing agent, impairing microtubule polymerization which is critical for cancer cell division 4 6 . Metabolic interference:  It inhibits glucose uptake by downregulating GLUT1 transporters and hexokinase II (HKII), key components of glycolysis, thereby starving cancer cells of energy and reversing drug resistance 1 4 6 . Activation of p53 pathway:  Fenbendazole induces mitochondrial translocation of p53, promoting apoptosis and cell cycle arrest at the G2/M phase 1 2 4 . Induction of multiple cell death pathways:  It triggers apoptosis, necrosis, autophagy, and ferroptosis, including ...
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